It is also pretty clear in the following excerpt from a study by the University of Hamburg that BHT is tumour promoting.
“Specific toxic effects to the lung have been observed with BHT…. BHT induces liver tumours in long-term experiments. Because there is no indication of genotoxicity of BHT, all published findings agree with the fact that BHA and BHT are tumour promoters. In contrast to BHA and BHT, vitamin E is not carcinogenic. On the other hand, all three antioxidants have also anticarcinogenic properties. The intake of the necessary high doses as for these effects are, however, contraindicated with BHA and BHT because of their carcinogenic effects.”
There are many more studies along similar lines, which link BHT to accumulating toxicity in the liver, lungs and thyroid, even adversely affecting blood coagulation. You can dig deeper by looking at the studies listed here.
After looking through that body of evidence it is a surprise to learn that studies are showing that BHT does not have an adverse impact, and may even have an anti carcinogenic effect of BHT on the body. They suggest at the least that BHT may be safe to ingest at low levels.
This began in 1979, when The National Institute for Cancer Research published a report stating that BHT was not carcinogenic.
A study conducted by Takahashi in 1993 found that BHT had virtually no effect on rats that were fed BHT orally over a number of generations.
Three generation toxicity study of butylated hydroxytoluene administered to mice. The dose levels of BHT in this study showed little adverse effect on reproductive and neurobehavioural parameters on mice.
Shockingly, a study conducted by Williams in 1991 suggested that there may be positive benefits to ingesting BHT.
Butylated hydroxytoluene lacks the activity of phenobarbital in enhancing diethylnitrosamine-induced mouse liver carcinogenesis.
These results suggest that the chemoprevention by BHT of cancer resulting from low-level long-term carcinogen exposure may be achieved at doses that do not produce adverse effects.
A Taiwanese study followed up in 1999, with the first demonstration that synthetic phenolic antioxidants decrease the N-acetylation of carcinogens and formation of DNA-carcinogen.
Copyright © 2002-2013. All rights reserved